Originally posted by Manu:
Mainstream scientists, however, say the evidence is irrefutable. HIV can be found in the blood of almost 100 percent of those diagnosed with epidemic AIDS, and virtually no one without HIV will develop AIDS.


See you know what bothers me here? "HIV Can be found in the blood of ALMOST 100% of those with AIDS... like... what about the ones that dont? Doesnt that point towards another sign?

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"It is easier to fight for one's principles than to live up to them."
Alfred Adler

Okay, my thing is this . . . and I HAVEN'T researched this enough, but I'm going with the logic I have.

We CAN test for an HIV virus, right? Okay, we KNOW it exists. We also know that "almost" all AIDs patients test positive for this virus. Almost IS a scary word IF it means 70-80%. BUT, if almost means 99.956%, then most should simply be replaced by the letter "l". I want to know numerically what "almost all" is.

The "non-HIV" people claim that AIDs is caused by drugs/malnutrition. Um-kay, maybe so . . . but where was aids before the 70's. Was it masked as other things? Maybe, but I doubt it. I think something like x% of the population getting sick and dying from common ailments would have raised a flag in the 50's. Surely we've had malnutrition and drug use longer than 1981?

Okay, and I'm NOT picking on gay men, but, if AIDs is truly a "generic" disease, why did it first attack gay men who had a sexual history of multiple partners? They can trace the "disease" by sex partners. They can even trace propagation. There isn't any evidence of the disease being passed any other way?

Has anyone just simply died of AIDs with a verifable history of no sex, drugs, or other risk factors. Has a 50 year old spinster developed the disease? Better yet, name a Hollywood star who WASN'T gay and died of AIDs? I don't know of ANYONE?

How do you explain all the hemophilliacs getting AIDs? The blood WAS tainted, and the things that killed these people weren't the normal diseases/conditions that attack people with this awful condition.

Okay, this woman hasn't developed AIDs, yet. There's ONE. I KNOW there are more, but how many HAVE developed AIDs and died?

See, the logic is based SOMEWHAT on fact but more on denial of a problem. Basically, if she can convince herself that she's fine, maybe the disease won't develop. But, basing her beliefs on "it doesn't happen to everyone . . . " is fool-hardy.

To me, it's like this. A woman in Russia survived a 7 mile fall from an airplane last year. This was VERIFIED and witnessed by 30-40 people. She didn't do WELL, but she is alive and awake. Now, even though SHE survived, I would still consider falling 7 miles to be fatal even though I can honestly say that only "almost" all people die from falling from that height.

For what it's worth, I DID have a friend die from AIDs. He was an athlete. He didn't drink. He ate WELL (his mother was a cook straight from heaven). He DIDN'T use drugs. We suspect (know) he was gay. He died . . . age 30.

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Go ahead and call Cosmo "Chief" and Bill is "Fearless Leader" . . . I'm HAPPILY "Minister of Spanking"!!

Famous Last Words:
Socrates - "I drank WHAT?"

I think people may be jumping the gun WAY WAY too early on saying that AIDS is merely 'common things.' I think a safer first step is to separate HIV/AIDS.

It could be that AIDs is a sexually transmitted disease that is SEPARATE from HIV. But then WHAT is HIV? What does IT do?

The other thing, you're rihgt about 'i'. Even if I is 90% that is scary.

BUT AIDs is a retrovirus. It has many forms and is constantly changing, perhaps HIV is?

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Manu Narayan

Her sight is the one I posted a link to on Stangnet a few months ago in the Magic Johnson thread. Not suprisingly I was flamed hard by all the simpletons over there.
Christine Maggiore knows her stuff, and she is right, someday the truth about this will come out, I believe that. There are people that cure themselves of "AIDS" just by moving to a different country where the definitions of the disease are different.


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AIDS is neither an infectious disease nor is sexually transmitted

by Roberto A. Giraldo, MD.

Note: The information on this website is presented for educational purposes and
is not a substitute for the advice of and treatment by a qualified professional.

This document was provided by
Continuum Magazine
VOL. 5 No. 3 www.continuummagazine.org (http://www.continuummagazine.org)

Roberto Giraldo is a Specialist in Internal Medicine from the University of Antioquia, Colombia. He graduated with distinction from the University of London after obtaining a MSc in Clinical Tropical Medicine. For 30 years he has been dedicated to clinical, academic and research activities in infectious diseases in Colombia, USA and Europe. He currently works in the Clinical Immunology section of the Department of Microbiology, University Hospital, New York City. He has been an independent researcher into AIDS for the past 15 years.

There are many scientific facts which show that the so-called human immunodeficiency virus (‘HIV’) does not fulfil the epidemiological and biological requirements, nor the common sense requirements, to be the cause of the human immunodeficiency syndrome (AIDS). 9-14, 26, 31-33, 36, 37, 44, 52, 59

‘HIV’ is neither necessary nor sufficient to cause AIDS, and antibody positivity does not always precede the development of the syndrome.11,32 This is demonstrated by thousands of AIDS cases that are ‘HIV’ negative and a host of people that are absolutely healthy and have never developed AIDS, even though they are diagnosed HIV positive. 2,13,20,31,32,49 HIV is not a pathogenic agent, and for this reason it cannot explain the immunological alterations, nor the pathogenesis, nor the natural history, nor the different clinical forms within the groups of people that develop AIDS. 9,13,14,29,30,32,41,42,59 What is called HIV has never been isolated as an independent, free viral entity.60 There are facts that question the existence of HIV as a real virus. 44

Since it has never been proven that ‘HIV is the cause of AIDS’, investigators who enthusiastically defend HIV as the cause of the syndrome have proposed a vast variety of agents as helpers or "co-factors" of HIV in the genesis of AIDS.21,48 However, these "co-factors" are by themselves causal agents of immunodeficiency and may generate AIDS with or without the diagnosis of ‘HIV’.12- 14,32,59 I prefer to call the "co-factors" immunological stressor agents.34

The new real circumstance that surrounds all the groups of people that develop AIDS with the greatest frequency is the exaggerated exposure in the last decades to a variety of stressor agents against the immune system, that can have a chemical, physical, biological, mental or nutritional origin.12,28,29

Coincidentally AIDS appears in various and distant groups of people in the second half of the twentieth century, at the time when the immune system of human beings is already saturated and has seriously deteriorated, due to involuntary exposure (and many times voluntary) to immunological stressors.28,32 The capabilities and functions of the immune system are neither infallible nor infinite. They have limits. The increment of stressors in the human ecosystem is putting in serious danger the preservation of our own species.28,34 AIDS is an alarm sounding.

The distribution of these stressors varies within the groups of people that develop the syndrome and this fact is the explanation for the different clinical forms of AIDS that occur in these groups.30,32 The immunological stressor agents create immunotoxic or immunogenic effects, or both, which generate a state of oxidative stress on immunocompetent cells and metabolic reactions of the immune system.29,56,57 Stressor agents also generate oxidative stress on other body systems.29,34 Progressive and continuous deterioration of the immune system causes a deficit of the defence, surveillance and homeostasis immunological functions, with the subsequent development of infections, neoplasias, and metabolic alterations.29,30 The severe weakening of the immune system and of the entire body eventually causes death.30 By contrast, all the definitions for AIDS created by the Centres for Disease Control and Prevention (CDC) are subjective, arbitrary, and include other less severe immunodeficiencies that are not AIDS at all.6,7

This conception of toxic pathogenesis and of the natural history of AIDS allows new forms of treatment and prevention that have positive repercussions on individual and community health.13,30

Drug treatments like AZT, the protease inhibitors and other similar antiretrovirals, must be eliminated from the treatment and prevention of AIDS, because they are immunotoxic agents and rather than producing wellness, they can generate AIDS.15,32,33,46,47

The prevention, control, and eradication of AIDS are easily possible and they depend on avoiding exposures to immunological stressors.12,13,30,32 The current programmes for preventing AIDS, based mostly on what is called "safe sex", with generalised and indiscriminate distribution of condoms, rather than achieving any benefit promote the risks of promiscuity, a potentially toxic lifestyle that helps undermine the immune system. 32,34,62 In the same way, the programmes of providing free "clean syringes" ("without HIV") to drug addicts stimulate addiction to drugs and indirectly promote the traffic of drugs.13,32 All the psychoactive drugs that are introduced to the body are potent immunotoxic agents.12,34

This toxic hypothesis of AIDS solves the problems that the infectious hypothesis [HIV/AIDS] has not yet solved, not to mention the millions of dollars invested in research, prevention, and patient care within the infectious conception of the syndrome.9-12,32,33

The so-called ‘AIDS test’ is neither sensible nor specific for detecting past or present infection with an HIV.39,41,42,49,58

Without reason it is used for diagnosis, or to decide the medications to treat or prevent this syndrome.2,38,41,42,49,58

‘HIV antibody’ positivity may act as a marker for immmunodeficiency, but is not generative of AIDS.13,16,56,57 HIV on the contrary could be an effect of the pathogenesis of this syndrome.29,31 There is scientific evidence that suggests that stressors of the cells of all species can work as inductor agents of viruses and virus-like parti-cles.5,8,25,31,32,43,51,54,61,68

The error over the etiology of AIDS was committed in part due to microbiologic prejudice in the mind of researchers, health professionals, journalists, and the public at large.31 This prejudice comes from the exaggeration of the germ theory of disease promulgated by Pasteur and Koch, which brought many benefits to the medical field at the time. Unfortunately, today they continue to think as at the end of the last century - that all is infectious, that all is contagious, and that it should be a microbe that causes everything. The world was prepared by a century of panic over microbes to mistake the etiology of AIDS. It was not possible to avoid it.

Another contribution to the error about the cause of AIDS is the failure in research methodology to fulfil epidemiological requirements.1,3,4,17-19,23,24,35,40,45,50,53,55,63-67,69-75 None of the postulates on which the infectious hypothesis of AIDS is based fulfil the requirements of the research method.2,9-14,27-34,56-59 None of the bases of the HIV-AIDS hypothesis has been demonstrated at an objective level.2,9-14,27-34,56-59 They are theoretical assumptions, created by the minds of those who generate and defend that hypothesis.22,38,48 Practically the entire world has become accustomed to believe all that we are told by the so-called men of science. Currently, the critical and questioning capabilities of ‘the people’ are null. They do not ask for the necessary proofs for the affirmations that can look objective.31 The worst epidemic that the contemporary world suffers is an epidemic of crises in the scientific method.27 It is more extensive than the AIDS epidemic. There will be more consequences unless we take a pathway paved with an authentic objective research methodology.

The scientific community has been wrong many times in this century, by considering as infectious diseases that are not -pellagra, scurvy and beriberi.14,31 The error currently made with AIDS has a larger magnitude due to the catastrophic repercussions on thousands of people that suffer from this toxic syndrome.32,33 Guilt for the error made with AIDS falls on a few researchers and health institutions of the United States government. The majority of people in the world simply believed the so-called men of science.

Analysis, understanding and solution of the error will force international medical authorities to rediscuss their tactics and strategies in the health care of people. This will lead to questions, investigations and solutions to the unfair forms by which men socially relate amongst themselves in modern society, which in the end are the reason for the existence of AIDS.

Let us go back to Hippocratic medicine. Let us divulge and stimulate the discussion about the cause of AIDS.

References:

1. Abramson JH. Making Sense of Associations. Factors and Risk Markers. Causes and Effects. In: Making Sense of Data; A Self-Instruction Manual on the Interpretation of Epidemiological Data. New York: Oxford University Press, 1988: 193-264, 219-228 y 265-316.

2. Alfonso HS. El Porque del Fiasco. In: El Gran Fiasco: El Sida no es Causado por el VIH. Barranquilla: Prestigio Editorial Colombiana, Distribution Universidad Metropolitana. 1996: 149-163.

3. Bratford-Hill AB. The Environment and Disease Association or Causation? Proc Royal Soc Med 1965; 58:295-300.

4. Buck C, Llopis A, Najera E, et al. Etiologic Investigations. Studies in Epidemics. In: The Challenge of Epidemiology; Issues and Selected Readings. Pan American Health Organisation, Scientific Publication No.505. PAHO, Pan American Sanitary Bureau, Regional Office of the WHO. Washington DC, 1988: 147-166 & 415-482.

5. Burnet FM. Virus as Organism. Evolution and Ecological Aspects of Some Human Viral Diseases. (Dunham Lectures, Harvard University, 1944). Cambridge, Mass.: Harvard University Press 1945.

6. CDC. Revision of the CDC Surveillance Case Definition for Acqured Immunodeficiency Syndrome JAMA 1987;258:1143-1154.

7. CDC. 1993 Revised Classification System for HIV Infection and Expanded Surveillance Case Definition for AIDS Among Adilescents and Adults. MMWR 1992; 41:1-19.

8. Dale HH. The Biologic Nature of Viruses. Nature (London) 1931; 128:599-602.

9. Duesberg PH. Retroviruses as Carcinogens and Pathogens: Expectations and Reality. Cancer Research 1987; 47:1199-1220.

10. Duesberg PH. Human Immunodeficiency Virus and Acquired Immunodeficiency Syndrome: Correlation but not Causation. Proc Natl Acad Sci USA 1989;86:755-764.

11. Duesberg PH. AIDS Epidemiology: Inconsistencies with HIV and with Infectious Diseases. Pro Natl Acad Sci USA 1991; 88:1575-1579.

12. Duesberg PH. AIDS Acquired by Drug Consumption and other Noncontagious Risk Factors. Pharm Ther 1992; 55:201-227.

13. Duesberg PH. How Much Longer Can We Afford the AIDS Virus Monopoly? In: AIDS: Virus or Drug Induced? Dordrecht: Kluwer Academic Publishers, 1996: 241-270.

14. Duesberg PH. Infectious AIDS; Have We Been Misled? Berkeley, CA: North Atlantic Books, 1996: 582.

15. Duesberg PH. ‘With Therapies Like This Who Needs Disease?’ In: Inventing the AIDS Virus. Forword by Nobel Laureate Kary Mullis. Washington, DC: Regenery Publishing, Inc. 1996: 299-359.

16. Ellison BJ & Duesberg PH. ‘Why We Will Never Win the War on AIDS’. El Cerrito, CA: Inside Story Communications, 1994:292.

17. Elwood JM. The Diagnosis of Causation. In: Causal Relationships in Medicine. A Practical System for Critical Appraisal. New York: Oxford University Press, 1988:163-182.

18. Enterline PE,. Sorting Out Multiple Causal Factors in Individual Cases. In: Chiazze L, Lundin FE, Watkins D. Methods and Issues in Occupational and Environmental Epidemiology. Ann Arbor Science, The Butterworth Group, 1983: 177-184.

19. Evans AS. Epidemiological Concepts and Methods. In: Viral Infections of Humans Epidemiology and Control. New York: Plenum Press, 1989: 1-32.

20. Fauci AS. CD4 T-Lymphocytopenia Without HIV Infection - No Lights, No Camera, Just Facts. NEJM 1993; 328: 429-431.

21. Fauci AS. Immunopathogenesis of HIV Infection. J Acq Imm Syndromes 1993, 6:655-662.

22. Fields BN. Time to Turn to Basic Science. Nature (London) 1994; 369: 95-96

23. Fletcher RH, Fletcher SW, Wagner EH. Risk. Cause. In: Clinical Epidemiology: The Essentials. Baltimore: Williams and Wilkins, 1996: 94-110 y 228-248.

24. Friedman GD. Making Sense out of Statistical Associations. In: Primer of Epidemiology. New York: McGraw-Hill Inc., 1994: 194-224.

25. Gibbs A. Molecular Evolution of Viruses: "Threes", "Clocks" and "Modules". J Cell Sci 1987; 7:s319-s327.

26. Gibbons M. Otro Enforque sobre la Teroria del SIDA. Taken from ADVANCE for Medical Laboratory Professionals, March 21, 1994. Translated into Spanish by Natalia Velez and Silvia Casabianca. El Pequeno Periodico, Publication of the Art and Science Foundation. Year XII, No.45. Medellin, Colombia, 1995; October 8 and 9.

27. Giraldo RA. AIDS Spread: Scientific Proof Missing. Advance for Medical Laboratory Professionals 1994; 6 (32):4.

28. Giraldo RA. AIDS amd Stressors 1: Worldwide Rise of Immunological Stressors (Abstrct). Toxicology Letters Supplement 1/78. 1995: s34.

29 Giraldo RA. AIDS and Stressors 2: A Proposal for the Pathogenesis of AIDS (Astract). Toxicology Letters Supplement 1/78. 1995: s34.

30. Giraldo RA. AIDS and Stressors 3: A Proposal for the Natural History of AIDS (Abstract). Toxicology Letters Supplement 1/78. 1995: s35.

31. Giraldo RA. AIDS and Stressors 4: The Real Meaning of HIV (Abstract). Toxicology Letters Supplement 1/78. 1995: s35.

32. Giraldo RA. Polemica Cientifica International Acera de la Causa del SIDA. Investigation y Educacion en Enfermeria (University of Antioquia, Colombia) 1996; 14:55-74.

33. Giraldo RA. La Indusrtia del SIDA: Manipulacion de un Error Cientifico. "El Pequeno Perodico", Puyblication of the Art and Science Foundation. Year Ano XIV, No.48, Medelin, Colombia, 1996; Nov: 8 and 9.

34. Giraldo RA. Papel de Estresantes Immunologicos en Immunodeficincia. Revista IATREIA (University of Antioquia, Colombia). Aproved to be published in July 1997.

35. Gordis L. Estimating Risk: Is There An Association? From Association to Causation.: Deriving Inferences From Epidemiologic Studies. More on Causal Inferences: Bias, Confounding, and Interactions. In: Epidemiology. Philadelphia: W.B. Saunders Company, 1996141-154, 167-182 y 183-195.

36. Guerrero CA. Es el VIH la Causa del SIDA? Bogota, Colombia: Deslinde, Magazine of Cedetrabajo. No.15, 1994; April/May: 100-122.

37. Guerrero CA. Es el Virus de la Immunodeficiencia Humana (VIH), la Causa del DIDA? Controversia Cientifica, Etica, Social y Politica de la Enfermedad. Bogota: Universidad Nacional Editorial Cientifica, 1994.

38. Ho DD. Time to Hit HIV, Early and Hard. NEJM 1995; 333: 450-451.

39. Hodgkinson N. Science Fails the "AIDS Test". In: ‘AIDS: The Failure of Contemporary Science. How A Virus That Never Was Deceived the World’. London. Fourth Estate, 1996: 233-262.

40. Jekel JF, Elmore JG, Katz DL. The Study of Causation in Epidemiologic Investigations and Research. Assessment of Risk in Epidemiologic Studies. In: Epidemiology, Biostatistics and Preventive Medicine. Philadelphia: WB Saunders Company, 1996: 54-56 y 74-78.

41. Johnson C. Playing Russian Roulete in the Lab: Can You Really Trust The AIDS Test? New York: The HEAL Bulletin, Special Edition. 1993.

42. Johnson C. Factors Known to Cause False-Positive HIV Antibody Test Results; Zenger’s California, September 1996: Whose Antibodies are They Anyway? Continuum (London), September/October 1996; 4:5.

43. Kanki PL, Hopper JR, Essex M. The Origin of HIV-1 and HTLV-4, HIV-2. Ann NY Acad Sci 1987; 511:370-375.

44. Lanka S. Collective Fallacy. Rethinking HIV. Continuum (London) September/October 1996; 4: 19-20. No Viral Identification; no Cloning as Proof of Isolation. Continuum (London) February/March 1997; 4: 31-33.

45. Last JM. A Dictionary of Epidemiology. Third Edition. Association. Causality. Risk Factor. Pathogenesis/Etiology. New York. Oxford University Press, 1995: 8-9, 25-26, 148-149 & 122.

46. Lauritsen J. ‘Poison by Prescription: The AZT Story’. New York. Asklepios, 1990.

47. Lauritsen J.’ The AIDS War; Propaganda, Profiteering and Genocide from the Medical-Industrial Complex’. Nerw York: Asklepiuos, 1993: 480.

48. Levy J. Pathogenesis of Human Immunodeficienct Virus Infection. Microbiological Reviews 1993; 57: 183-298.

49. Maggiore C. ‘What if Everything You Thought You Knew About AIDS Was Wrong?’ Los Angeles: HEAL (Health Education AIDS Liaison), 1996:41.

50. Malenka DJ, Baron JA, Jhonson S, et al. The Framing Effect of Relative and Absolute Risk. J Gen Intern Med 1993; 8:543-548.

51. Mayr E. Driving Forces in Evolution; An Analysis of Natural Selection. In: Morse SS. ‘The Evolutionary Biology of Viruses’. New York. Raven Press, 1994: 29-48.

52. McDonald JF, Editor. Special Issue: Alternative AIDS Hypothesis. Genetica 1995; 95:1-202.

53. McMaster University Health Sciences Centre, Dept. of Clinical Epidemiology and Biostatistics. How to Read Clinical Journals IV; To Determine Etiology or Causation. Can Med Assoc J 1±981; 124:985-990.

54.Morse SS. Examining the Origins of Emerging Viruses. In: Emerging Viruses. New York. Oxford University Press, 1993: 10-29

55. National Conference on Cloustering of Health Events. Am J Epidemiol 1990; 132:s1-s202.

56. Papadopulos-Eleopulos E. Reappraisal of AIDS: Is the Oxidation caused by the Risk Factors the Primary Cause? Med Hypotheses 1988; 25:151-162

57. Papadopulos-Eleopulos E, Turner VF, Papadimitriou JM. Oxidative Stress, HIV and AIDS. Res. Immunol; 143: 145-148

58 Papadopulos-Eleopulos E, Turner VF, Papadimitriou JM. Is a Positive Western Blot Proof of HIV infection? Bio/Technology 1993; 11:696-707

59. Papadopulos-Eleopulos E, Turner VF, Papadimitriou JM et al. A Critical Analysis of the HIV-T4-Cell AIDS Hypothesis. Genetica 1995; 95:5-24

60. Papadopulos-Eleopulos E, Turner VF, Papadimitriou JM, Causer D. The Isolation of HIV: Has it Really Been Achieved? The Case Against. Continuum (London) September/October 1996; 4 (supplement): 1-24

61. Penny D. Molecular Evolution: Origins of the AIDS Virus. Nature (London) 1988;333:494

62. Root-Bernstein RS. ‘Rethinking AIDS; The Tragic Cost of a Premature Consensus’. New York: The Free Press, 1993:512

63. Rothman KJ. Causal Inference in Epidemiliogy. Multivariate Analysis. Interactions Between Causes. Analysis with Multiple Levels of Exposure. In: Modern Epidemiology. Boston: Little Brown, 1986: 7-22, 285-310, 311-326, 327-350.

64. Rothman KJ. Causal Inference. Chestnut Hill: MA; Epidemiology Resources, 1988: 207

65. Rothman KJ. Adjustments are Needed for Multiple Comparison. Epidemiology 1990; 1:43-46

66. Rothman KJ, Greenland S. Causation and Causal Inference. In: DetelsR, Holland WW, McEwen J, OmennGS. Oxford text book of PublicHealth. Third Edition. Vol 2; The Methods of Public Health. New York: Oxford Universiy Press, 1997: 617-630

67. Schlesselman JJ. "Proof" of Cause and Effect in Epidemiologi Studies: Criteria for Judgements. Prev Med 1987; 16: 195-210

68. Strauss EG, Strauss JH, Levine AJ. Virus Evolution. In: Fields NB, Knipe DM. Fundamental Virology. Second Edition. New York. Raven Press, 1991: 167-190

69. Susser M. Causal Thinking in the Health Sciences: Concepts and Strategies of Epidemiology. Oxford: Oxford University Press, 1973: 181

70. Susser M. Rules of Inference in Epidemiology. In: Regulatory Toxicology and Pharmacology. New York: Academic press, 1986: 116-128

71. Susser M. What is a Cause and How Do We Know One? A Grammar for Pragmatic Epidemiology. Amer J Epidemiol 1991;113:635-648

72. Vasco-Uribe A. Curso de Metodologia de la Investigacion en Salud. Modulo 4: La Causalidad. Barcelona: IDER S.L., 1993: 76 AIDS and stressors by Roberto A. Giraldo, MD

205 pages

ISBN 958 9458 03 3

Published by Fundación Arte y Ciencia, Medellin, Colombia




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Red 86 GT


"Fascism should more appropriately be called Corporatism, because it is a merger of State and corporate power."
Benito "Il Duce" Mussolini

Did Dr. Gallo and his Colleagues manipulate the "AIDS-Test" to order?
"The hunt for the virus"1 has degenerated into "clean torture with fatal results".2
Dr. med. Heinrich Kremer - Dr. Robert Gallo

Note: The information on this website is presented for educational purposes and
is not a substitute for the advice of and treatment by a qualified professional.

This document was provided by
Continuum Magazine
VOL. 5 No. 4 www.continuummagazine.org (http://www.continuummagazine.org)

Whoever, (for reasons given below) casts doubt on the theory that "HIV causes AIDS", is often confronted with the question, if it does not, how is it that a patient who has been diagnosed as "HIV positive" by the test sooner or later goes on to develop AIDS? To which the AIDS sceptic usually replies that a "HIV-positive" laboratory result, an arbitrary defined characteristic, is part of the clinical diagnosis "AIDS". This exchange does not advance the argument very much as to whether "AIDS" and "HIV" are scientifically-speaking biological entities and if between them a biological cause-effect relationship is possible. In other words, if either the term "AIDS" or the term "HIV", (or both), do not represent conceptually independent entities but rather semantic constructs, then biologically there can be no cause-and-effect relationship between these two terms, i.e. between the postulated pathogen "HIV" and the supposed defin-able disease entity "AIDS".

The causative factor, the "retrovirus HTLV-111" (termed "HIV" since 1987) was introduced by Robert Gallo in 1984 (then a retrovirologist in the Tumour Biology Laboratory in the National Cancer Institute at Bethesda). On May 4th, 1984 together with collaborators from his own laboratory and other research centres and hospitals as well as workers at the pharmaceutical company Litton Bionetics, he published four basic papers in Science. 3-6 These supposedly described the identification, isolation and continuous production of a newly discovered type of retrovirus as well as the serological analysis of this "HIV" and of tests capable of detecting antibodies to "HIV" in the sera of "patients with AIDS or pre-AIDS". The simultaneous publication of these four papers by Gallo et al was shortly preceded by a patent application for "HIV antibody tests" and by Reagan’s US Health Secretary’s announcement, at a press conference attended by Robert Gallo himself, before the world’s media that Robert Gallo and his team had "discovered the probable cause of AIDS".

The first Science paper of May 4th, 1984 begins with the fundamental assumption: "epidemiological data suggest that the acquired immunodeficiency syndrome (AIDS) is caused by an infectious agent that is horizontally transmitted by intimate contact or blood products".3 The word ‘probably’ employed by the US minister only a few days before was no longer mentioned by Gallo et al.

The fourth and last Science paper of that date ends with the conclusion: "The data presented here and in the accompanying reports suggest that HTLV-111 is the primary cause of AIDS" 6 . (HTLV-111 = HIV). Gallo et al’s conclusion proves that they did not postulate a direct cause-and-effect relationship between "HIV" and "AIDS", declaring "HIV" to be only the primary cause of "AIDS": "Although the disease is manifested by opportunistic infections, predominantly Pneumocystis Carinii Pneumonia, and by Kaposi’s Sarcoma, the underlying disorder affects the patient’s cell-mediated immunity, resulting in absolute lymphopenia and reduced subpopulation of helper T lymphocytes (OKT4+)".3 Gallo et al by no means, therefore, postulated that "HIV" was the direct cause of "AIDS"; rather, they only claimed "HIV" is the cause of "AID" (AID = Acquired ImmunoDeficiency = reduced sub-population of T-helper lymphocytes). The syndrome "S" ("manifested by opportunistic infections (OI), mainly Pneumocystis Carinii Pneumonia (= PCP), and Kaposi’s Sarcoma (= KS)") was presented by Gallo et al as if automatically the necessary consequence of "AID".

The scheme of Gallo et al is as follows:
1. "HIV" causes "AID", as a consequence of the infection and sooner or later the destruction of T-helper lymphocytes.
2. As a consequence of the decrease of cellular immunity, the control of opportunistic pathogens and cancer cells by T-helper lymphocytes breaks down as a result of which, syndrome "S" develops.

The short version of Gallo et al’s plague formula is "HIV =AID = S".

The two part causal chain "HIV causes AIDS" actually therefore turns out to consist of three parts, and Gallo et al’s claim that "HTLV-111" (= "HIV") is the primary cause of "AIDS"6 is a fusion of two hypothetical causal assertions, and a fictitious end-effect assertion. This is because Gallo et al’s published data say nothing about whether "AID" really does cause "S"; they can at most suggest a cause-and-effect relationship between "HIV" and "AID". Whether "S" can be the result of "AID" is for several reasons highly doubtful. "S" is somewhat chameleon-like due to numerous re-definitions undergone, so that the existence of "S" as a "separate disease entity",4 in the sense of a biological disease entity, can no longer be rationally made out. Individual, defined diseases which initially made up part of the syndrome were years later expressly removed again. In the end a wild collection of 29 old infections and non-infectious diseases has been put together to constitute the syndrome "S", of which several are part of "S" even if the "HIV" status is negative or indeterminate.7

The latter means that "AID" cannot be the cause of "S" because "AID" is supposed to be the result of "HIV", in order that Gallo et al’s plague formula "HIV = AID = S" as a causal chain is upheld, yet "AID" due to different reasons can exist independently of "HIV". Nothing is given whereby "AID" must be the only cause of "S". "AID" and "S" could, instead, have a common cause which need have no causal relationship with a hypothetical "retrovirus HIV".

The pretence of a pseudo-biological cause-and-effect relationship expressed by the plague formula "HIV = AID = S" has made a leading AIDS critic, who has presented the most comprehensive clinical analysis of the AIDS phenomenon, say, "AIDS, in short, has become a schizophrenic disease".8

How then, can a semantic construct of a collection of mostly contradictory diseases be the result of a supposed biological causal chain, which itself in turn is made up of hypothetical constructs as cause-and effect factors? Because the premises and conclusions 3,6 which underlie Gallo et al’s plague formula can be falsified convincingly.

Gallo et al have claimed that epidemiological data prove that an infectious agent 3 is the cause of "AID", and "AID" is the cause of "S". Essentially, Gallo et al arrived at this conclusion from the findings of the CDC that "S" ("OI, mainly PCP, and KS") is significantly connected with very frequent promiscuity and predominantly receptive anal intercourse in homosexual men in the metropolitan areas in the US.3 However, this conclusion only demonstrates the arbitrary and selective interpretation of the clinical data by the CDC and Gallo et al.

Highly promiscuous and predominantly receptive (unprotected) anal intercourse are specifically indicators simultaneously for infectious and non-infectious causal factors for "S" ("Ol, mainly PCP, and KS") as well as "AID" (decline in T-helper lymphocytes in blood serum). The conclusion of a new infectious pathogen and simultaneous exclusion of all non-infectious causal factors is by no means compelling, although it determines to this day the theory that "HIV causes AIDS".

Highly promiscuous behaviour and predominantly receptive anal intercourse closely correlate with consumption of sexual stimulants, above all amyl and isobutyl nitrites. 95% of homosexual men in the US report regular use of nitrite.9,10 Nitrite inhalation relaxes the smooth anal muscles, raises blood flow to the penis, raises pain threshold, heightens orgasm and unleashes a mild state of intoxication in the brain. Nitrite use predominantly but not exclusively became known in homosexual sex partners, and has been approaching ubiquitous in surveyed homosexual men in Western countries since the mid-70s.11,13

High frequency promiscuity and predominantly receptive anal intercourse very often entails concomitant increased multi-infectivity and provocation of administrating antimicrobials, chemotherapy, antibiotics, antiparasitica, antimycotica, virusstatica and corticosteroids.14 The first report by the CDC in June 1981 of five diseased homosexual men being treated for PCP contains some clinical information of their medical history and medication, because at the time, the all-encompassing description AIDS, masking the real symptoms, had not yet become entrenched: The five homosexual patients had not had sexual relations between themselves. All of the five patients used nitrites, and all five had been treated with TMP/SMX (TMP = trimetho-prim, SMX = sulfamethoxazole).15

The substances TMP/SMX, also known as bactrim and septrin were introduced in the early ’70s as a double chemotherapeutical folic acid inhibitor. Nitrite and SMX (a sulphonamide derivative) are strongly electrophilic oxidising agents. Both oxidise ferrous iron in haemoglobin to ferric, and thereby reduce oxygen-binding capacity of red blood cells. This causes methaemoglobulinaemia, 16,20 a progressively life-threatening deficiency in oxygen supply into the respiration chain of the mitochondria. The latter are former bacteria, which, as multifunctional organelles, supply energy to the whole cell in the form of adenosine triphosphate (ATP) produced in oxidative phosphorylation.21 Oxygen-dependent ATP synthesis and its resulting oxygen metabolites control the cell division cycle. If too little oxygen is transported to the respiratory chain, the ratio of oxidative ATP production in the respiration chain (normally about 90%) may become inverted in favour of the non-oxidative ATP production (normally about 10%). Latest experimental findings suggest that the redox balance controls the genetic expression of proteins for the enzymes of the non-oxidative ATP production (glycolysis).22

Under normal physiological conditions, there is a rhythm of phase-linked change between oxidative energy production in the mitochondria and non-oxidative glycolysis during the late stage of cell division (the S-phase of mitosis). If, through lack of oxygen under conditions of methaemoglobulinaemia, the genetic expression of glycolytic enzymes is not sufficiently inhibited,23 the cell may, despite intact mitochondria, and the presence of residual molecular oxygen, switch to permanent non-oxidative glycolysis and cationic load reversal. This results in unrestrained cell division, which may ultimately lead to transformation to a tumour cell.

Along the oxygen transport route in the bloodstream, conditions in the most minute capillaries with a diameter below 100 nanometres, because of altered partial pressure of oxygen, are particularly favourable for the oxidation of the red haemoglobin, which can only bind oxygen in its reduced form. Through diffusion and association to essential fatty acids through transit routes of the basic-tissues it can deliver oxygen to individual cells. The mechanism of unrestrained activation of cell division (hyperplasia) in methaemoglobulinaemia, may, therefore, following hypoxaemic stress, above all in the smallest capillaries, affect the cells of the capillary walls - the endothelial cells. These endothelial cells are in direct contact with the hypoxaemic red blood cells. If hyperplastic conversion of endothelial cells occurs, that is called Kaposi’s Sarcoma. On the other hand, especially in rapidly dividing cells such as in thymus-matured precursor cells of T-helper lymphocytes, ATP production can decline to a critical value, if oxygen turnover is reduced permanently even by a small amount. This is a control mechanism, which in turn may affect the rate of mitosis. This interaction of haemoglobin oxidation by nitrites and antimicrobial drugs with oxidative phosphorylation may, in a situation of increased simultaneous consumption of T-helper lymphocytes as a result of slowing maturation of T-helper lymphocytes, be in part a cause of "AID".

c l e a n t o r t u r e

This chain of causal events is also supported by the "frightening possibility" 24 that nitrites may turn most classes of antibiotics into carcinogens.25 Excessive antibiotic consumption (whether prescribed or not; in a study 40% of male homosexuals admitted preventive use 26 ) in conjunction with nitrites is a frequently encountered pattern of behaviour among male homosexuals especially in the large urban areas in Western countries.27

Hypoxaemic stress can, therefore, explain the contradiction of the simultaneous appearance of malignant hyperplasias (KS, lymphomas) and opportunistic infections, mainly PCP, in homosexual men (approx. 2/3 of "AIDS cases" in Western countries, excluding undeclared homosexual "AIDS patients" estimated by orthodox "AIDS"-doctors to amount to 50% of so-called heterosexual risk groups 28 ), without ever introducing a hypothetical "retroviral" cause to explain the pathophysiology.

In contrast to this clear finding, Gallo et al tried to resolve the clinical contradiction between OI and KS by constructing a new "retrovirus HIV". Gallo et al’s so-called retroviruses "HTLV-1" and "HTLV-11" are said to cause rare forms of leukaemia, i.e. cancers of the white blood cells, whereas "HTLV-III" (= "HIV") is said to kill T-helper lymphocytes.

This concept has completely failed. The cytopathic effects of "HIV" demonstrated by Gallo et al have turned out to be laboratory artefacts.29 Gallo et al’s claim that "HIV" kills T-helper lymphocytes could, despite changing the theories, not be confirmed.30-33

The disease theory "HIV causes AIDS" is itself based on several serious clinical misconceptions:
1. The agent causing PCP is not as Gallo claimed a protozoon. The aetiology according to which after the destruction of T-helper lymphocytes by "HIV-infection", Carinii pneumocytes, the cause of PCP, could escape control by T-helper lymphocytes and multiply unrestrictedly, is objectively wrong. Such protozoa simply do not exist.34,35 What is involved are micro-fungi that are inhaled in the air, and which, for example, in the case of increased cell decay following hypoxaemic metabolic changes (including "AIDS" without "HIV"), find fertile terrain in the alveoli of the lungs. In this way, a harmless fungus (saprophyte) becomes the dangerous cause of PCP.

2. Contrary to what Gallo et al claimed, T-helper lymphocytes do not suppress the growth of cancer cells, because cancer cells do not have antigens through which T-helper lymphocytes could identify them.36 This means that the hypothetical destru-tion of T-helper lymphocytes by "HIV" and the ensuing disappearance of the suppression of KS cells cannot be the cause of KS. The predicted increase of all other types of carcinoma in "AIDS patients" resulting from the disappearance of the surveillance of cancer cells after the postulated destruction of T-helper lymphocytes by "HIV-infection" did not occur.37

3. Contrary to the assumption of the CDC and Gallo, the hypothetical "HIV infection" of T-helper lymphocytes, despite the postulated essential alarm function of T-helper lymphocytes for antibody production by B-plasma cells, did not result in destroying defence capacity against all microbes. Unlike patients with impaired immune functions, e.g. intensive care patients in whom mortality following typical bacterial infections is up to 80%, strikingly in the "immune deficiency syndrome AIDS", bacterial infections are rarely seen. The CDC under the category "AIDS indicator diseases" states explicitly for "bacterial infections, frequent or repeated": "not applicable as indicator of AIDS in adults/adolescents".37

4. A fundamental pillar of the disease theory of Gallo et al according to which "HIV causes AIDS", is severely dented by the actual epidemiological situation over the 15 years 1982-1997. For example, in 1997 the German "AIDS Centre" registered 2736 KS cases in total with 2505 KS cases in the category "homosexuals". The remaining KS cases were in "heterosexual risk groups" or "no information on risk group". On average, therefore, there were 15 KS cases a year, which were not primarily classified as "homosexual". Because homosexual intravenous drug users are classified as intravenous drug users and at least 50% of the patients classified as "heterosexual men" and "not known" were subse-quently reclassified as homosexuals,28,38 this is of the order of magnitude to be expected for KS cases classified as "non-homosexual men". Corresponding epidemiological data for the prevalence of KS are available for other Western countries 39 .

Gallo et al’s formulation "HIV = AID = S" is not, therefore, found to be true. "AID" (measurable decline in lymphocyte population in the blood, especially T-helper lymphocytes) though it can occur, in all members of "high-risk groups", is evidently not the cause of "S" ("OI, mainly PCP, and KS") because "S" can, first, occur without "AID",29 and secondly, the combination of "S" (with KS) should, if the theory were correct, not exclusively be limited to homosexual patients. If, therefore, "S" is not necessarily the result of "AID", what then is the common pathogenic indicator of "AID" patients as defined by Gallo et al to be "high-risk groups"?4

The common factor of "AID" patients (without necessarily resulting in "S") is obviously the unusually high uptake of strongly oxidising substances (mitogens), and the huge variety of exogenous extraneous cells such as red blood cells, activated lymphocytes or sperm cells from individuals (allogenic stimulation.29,40 ) It is beyond doubt that this oxidative stress (i.e. pro-oxidative vs. anti-oxidative metabolism) of "high-risk groups", can overload the detoxification capacity and waste disposal capacity of the body which is furthermore supported by the finding that asymptomatic "HIV positives" belonging to "high-risk groups" show a strong shift from reduced to oxidised glutathione.41

The glutathione system is essential for the removal of oxygen free-radicals, especially in the mitochondria.42,43 The oxidation of the central molecule of glutathione, cysteine, to cystine, in a chain reaction reduces the build up of glutathione and accelerates the destruction. It follows that the systemic decline of glutathione concentration in HIV positives can be due to both decreased synthesis and increased disposal.

"The oxidative stress to which AIDS patients are subjected would lead to cellular anomalies in many cells, including lymphocytes, resulting in opportunistic infection, immunological abnormalities and neoplasia".44

Does this finding of the overload of redox potentials in members of "high-risk groups" mean that "HIV", too, or rather the "anti-HIV antibodies", are the result of oxidative bombardment on the cell-mediated immunity of the "high-risk groups"?

A specific load value of the diminution of the reduction force in the bodies of members of "high-risk groups" is hepatitis type B, in particular, in the chronically active form.45 Gallo et al postulated in the first paragraph of the first publications in Science of May 4th 1984 (except for the first rebutted premise: "Epidemiological data suggest that the acquired immun-odeficiency syndrome (AIDS) is caused by an infectious agent" and the second (rebutted) premise: "AID" necessarily leads to "S"), a third premise: "Although patients with AIDS or pre-AIDS are often chronically infected with cytomegalovirus or hepatitis B virus, for various reasons these appear to be opportunistic or coincidental infections".3

This claim stands the clinical history completely on its head. "High-risk groups", in Gallo’s definition , "homosexual men with multiple sex partners, intravenous drug missusers, haemophiliacs, blood transfusion recipients and close heterosexual contacts of members of these high-risk groups"6 were long before the so-called ‘sudden’ arrival of "HIV" (1978), recognised to be the most severely hepatitis-B affected groups of patients.46-50 Hepatitis inducers (nowadays thought to be hepatitis-B, hepatitis-C) "appear to be thousands of times as infectious in clinical settings as HIV and represent a much more prevalent medical problem".51 Hepatitis-B due to various patho-physiological reasons, especially in the chronically active form, contributes significantly to oxidative stress, by restricting waste disposal and detoxification, and overloading redox potentials. The body tries to compensate for this by increasing cortisol production. When this ultimately fails, hypercorticolism persists in a damaging way. A hypercatabolic metabolism results from this (i.e. excess cell decay vs. build up).52 Cortisol as "synergiser" for a number of hormones and mediators effects activation of cyclic adenosine monophosphate (CAMP) and a displacement of the cAMP/cGMP ratio as principal indicator for increased cell turnover.53 The net effect is a dampening of cellular immunity and activation of humoral immunity. Resulting from the increased cell turnover, the decreased disposal of cell debris (because of the dampened cellular immunity, "AID") and the strengthened autoimmune activity, a significantly increased formation of autoantibodies occurs which above all specifically bind to cytoskeletal proteins and extra-cellular proteins of the cell matrix as antigens.54,33

In conclusion, it is fair to assume that Gallo et al took these attributes 25 of "high-risk groups" into consideration, namely,
1. the excessive oxidative (mitogenic) stress
2. allogenic stimulation by foreign cell components
3. the sharply increased antigen auto-antibody load together with suppression of T-cell dependent immunity brought about by synergistic effects of persistent corticolism with resulting change in cAMP/cGMP ratio.

f a t a l r e s u l t s

In their original paper ("Detection, isolation and continuous production.."),3 Gallo et al were able only to cite indirect phenomena, such as reverse transcription, ultra-thin layer electron micrographs, banding of protein mixtures at given densities, which according to the established rules of virology are not acceptable as evidence for the existence of a virus and even less a "retrovirus", because these indirect phenomena can also be obtained in the absence of any viral entity under certain cell culture conditions.55-60,33

Then the question becomes increasingly pressing: how did Gallo et al manage to produce a protein mixture in cell cultures and in the test tube, which, as the substrate for the "AIDS-test" when in contact with serum of people in "high-risk groups", resulted in a given rate of antigen antibody-reaction for single proteins?6

Gallo’s papers, though written in highly technical language, do not reveal this secret of test-constructing. Only in 1987 when the disease theory "HIV causes AIDS" led to the introduction of a highly toxic DNA chain terminator (azidothymidine = AZT = Retrovir), was some light shed on this matter when two of Gallo’s former collaborators and co-authors of the original publications in Science of May 4th 1984 3-6 revealed the essential details. Mangalasseril Samgadharan and Phillip Markham (collaborators at Litton Bionetics, Kensington MD, USA) published the biochemical methods used by Gallo et al whereby they manipulated the protein mixture which due to self-defined conventions is said to be "HIV antigens".59

To start with, Gallo et al biochemically prepared cell compo-nents obtained from members of "high-risk groups" according to the self-defined rules of "retrovirus production". This procedure, only "from time to time" and only transiently,61 led to the production of unspecific phenomena as surrogates for the existence of a new "retrovirus". Then they mixed lymphocytes from patients in "high-risk groups" with exceptionally rapidly dividing leukaemia cells.3,4 This cell mixture was then subjected to the effects of certain biochemical substances. They go on to say that "in vitro stimulation was achieved by mitogens or added cells (allogenic antigens )...Certain manipulation of culture conditions improved the result, for example, co-cultivation of patients’ cells with peripheral white blood cells, which were stimulated by mitogens, from non-infected donors."

The "virus isolation" of cultured cells was also significantly facilitated by adding hydrocortisone to the culture medium".61

Knowing the specific antigen auto-antibody status of "high-risk groups" patients, it is possible, therefore, to trigger, on demand, an antigen mixture appropriate to the auto-antibody repertoire in serum from high-risk patients, in cell cultures of human lymphocytes, co-cultured with leukaemic cells when subjected to specific biochemical manipulation.

The apparent proof that in the antigen mixture one is dealing with "retroviral" proteins - brought about by the demonstration of a naturally occurring repair mechanism, reverse transcriptases, produced particularly copiously in cancer cell cultures to repair DNA and renew chromosome ends, hence cocultivation with leukaemic cells in Gallo et al cell culture 3,4 , as well as proof of exocytotic virus-like particles (frequently occurring transport particles to expel intra-cellular components from mitogenically stimulated cells) as proof of "isolation and continuous produ-tion" of supposed retroviruses, is misinterpretation.33

That Gallo et al’s sensational discovery of a "new retrovirus" was in fact a laboratory artefact is made explicit by Gallo et al’s expressly stating that "HTLV-1" (isolated from T-cells in 10% of "AIDS patients") and "HTLV-11" from the "family of retro-viruses" in "AIDS patients", were also discovered and demon-strated. 3,4 Later on, there was no further mention of "HTLV-1" and "HTLV-11" being "isolated from T-cells of AIDS patients". Nor were there noticeable occurrences of leukaemia in "AIDS patients". The "isolation" of "HTLV-1" and "HTLV-11" was a laboratory artefact due to the rules of "retrovirus production" of Gallo et al. By analogy this finding accounts for "HTLV-111" (= "HIV") as well.

In effect, therefore, Gallo et al were adapting conditions which they knew to be conducive to antigen formation in the body of "high-risk patients", to laboratory conditions. The difference is that in cell culture as opposed to the body of "high-risk patients", no antibodies are present because the B-plasma cells are absent. Then it is possible, at a certain arbitrarily fixed auto-antibody level, to demonstrate an antigen-antibody reaction when the antigen mixture of the cell culture is brought in contact with sera of "high-risk patients". This is exactly the principle employed in "HIV-antibody tests". In mirror image fashion, the artificially produced antigens bind to the auto-antibodies, whose presence was to be expected because of the well-known patho-physiological overload of "high-risk patients".

In describing the recipes of Gallo et al, who covered their laboratory-tricks behind the dust screen of patents, the irrational reduction of "AID" to the effect of a seemingly new infectious cause 3 and the ignoring of the clinical effect of chronic hepatitis 3 becomes apparent as a claim used to create pressure to introduce the patented "antibody test system" of a "new retrovirus" found in the National Institute of Cancer.

The laboratory finding of "HIV positive" which may be diagnosed in those belonging to "high-risk groups" depending on the quantity and personal reaction pattern of antibodies, may also be made in rare cases in those not belonging to "high-risk groups" for a number of extremely diverse reasons.

Gallo et al’s expectations regarding the dynamics of the spread of "HIV" have, contrary to the horrendous predictions, not been fulfilled in the real biological world. In Germany, for example, according to official figures for the 15 years 1982-97, out of a population of 82 million, 60.000 have been notified as HIV positive, i.e. more than 99.9% of the population are personally not affected by "HIV" and "AIDS". The official government forecasts, until now uncontradicted, spoke of there being more "AIDS cases" by 1996 than there were inhabitants. At least every other person was supposed to have died by 1996, unless a vaccine or drug against the "absolutely" fatal plague had become available60 In the former East Germany, there have been a grand total of 252 cases in a population of 16 million, and that despite massive migrations (since the fall of the wall) up to the end of 1996. Over the past decade in the whole of Germany there has been a very constant 2-3,000 people diagnosed annually as "HIV positive". 95% of these have been classified as belonging to the "high-risk groups" of "homosexual men" and "IV-drug users" (homosexual IV-drug users are counted as ordinary IV-drug users). 5% of "HIV positives" are considered to be false positives, but cannot be identified as such by the test.

At most 2000 "HIV positives" develop AIDS annually, and 1300 patients die annually of "AIDS" (actual cause of death is not revealed). Of the supposed 60,000 HIV positives (figures are very unreliable because of unknown multiple reporting), 50,000 are still officially alive today. 54% of all "AIDS patients" gave their addresses to be one of the six largest cities, in which 10% of the general population also live. Opposed to that in 90% of the remaining inhabitants only 44% of the notified "AIDS cases" occur.

By ay of example, the disease rate and death rate of "HIV-positive" haemopholiacs registered in these six cities is twice as high as in "HIV-positive" haemophiliacs living outside of those cities. In these cities (Berlin, Hamburg, Köln, Düsseldorf, Frankfurt and München) the university clinical "AIDS-treatment centres" are located, which report the highest "AIDS"-disease and death-rates to the national AIDS-centre. As the positions of collaborators in the "AIDS-ambulances" and "AIDS-stations" of these university clinics mostly are paid for by the pharmaceutical companies, the connection between Medicine and the markets ("AIDS-test", "AIDS-medications") becomes all too obvious. Very intriguing is the comparison between the "capitalist" West-Berlin and the former "socialist" East-Berlin.

In the period of 15 years from 1.1.1982 to the 1.1.1997 in West-Berlin (2.2 million inhabitants, which make less then 3% of Germany’s population), 3083 "AIDS-cases" have been registered which are 20% of all German "AIDS-cases". In the same period (including 7 years of unification with West-Berlin after the fall of the Berlin Wall 1989) in East-Berlin (1.3 million inhabitants = 1.6% of the German population) only 152 "AIDS-cases" are registered, which make 1% of all German "AIDS-cases". This very intriguing, chance, historical and model-like data 38 , proves wrong the premise of Gallo et al. that "epidemiologic data suggest that the acquired immunedeficiency syndrome (AIDS) is caused by an infectious agent". The disease rate when brought in connection with the whole population is obviously a very rare medical event, not dependent on a ubiquitous transmittable mass-virus, but determined by life-style in a largely commercialised subculture and/or by uncritical medical intervention in Western societies of superabundance.

Or patho-physiologically speaking: "AIDS-patients" fall ill due to a lack of power of reduction (caused by superoxidation and/or hypoxaemia) in the midst of a redundant medical over-supply.

Arguing against this, Gallo et al. refer to Africa, which is uncritically presented by mass-media as the "dying AIDS-continent". In this context too the world of facts is seemingly overwhelmed by a virtual world of imaginary information.

In Africa south of the Sahara, the annual increase in population was about 100 million inhabitants over the last decade, even though the latest report on the world population states that according to a lot of population experts "in the third world the plague supported birth-planning more than any earlier programs".63 Due to lack of medical infrastructure and low budgets in the health care system (in most states south of Sahara the average annual spending per head of the population for providing health care is US$6: a single complete "AIDS-test" - 2 x ELISA-test, 1 x Western blot, costs much more than 6 US$), the "AIDS-test" is not widely used. Instead the World Health Organisation (WHO) transfers certain amounts of money to the health authorities of the various countries for "AIDS-education" in order to get estimated incidental rates of "HIV-infection" and "AIDS-cases" which are not verified by the WHO.

WHO-experts use these estimates in calculations based on the supposed "dynamic of distribution" of the "HIV-plague" and present the resulting numbers to the world media as "HIV-infection" and "AIDS-disease" in Africa. Usually, in the subsequent media reports the speculative "HIV-infections" and "AIDS-diseases" are lump-summed and wrongly reported as "AIDS-cases" in Africa. This is the way the manipulated numbers of more than 20 million "AIDS-cases" in Africa (app. 90% of the world-wide reported "AIDS-cases") came into existence without any substantial base of knowledge.64

Thus the fictitious looming scene of a "people murdering AIDS-plague" in the "global media village" enhances sales of "AIDS-tests" and "Anti-HIV-medications" (euphemicly termed "cocktail therapy") in western countries, using "poor Africa" to increase sales in the "rich West".

The data on the clinical, immunological, virological and epidemiological progress since 1984 show beyond doubt that the disease-theory "HIV causes AIDS" has no concurrence with the biological reality. As a marketing strategy Gallo’s manipulated "AIDS-test" has been extremely successful. But this at the cost of the health and life of uncounted children, women and men who, from a medical ethics point of view became victims of "clean torture leading to death" induced by the arbitrary medical death sentence of a "HIV-positive" result. Medical ethical behaviour "according to best wisdom and conscience" must require, within one’s own responsibility, the effort to inform oneself on the basis of existing data about possible manipulations in diagnostic tests and therapy, and to use appropriate alternative therapies instead of inducing fear blind with rage.33

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44 Papadopulos-Eleopulos E, Turner VF, Papadimitrou JM. Oxidative stress, HIV and AIDS. Res Immunol 1992;143:145-148.

45 Hässig A, Kremer H, Liang WX, Stampfil K. Parenteral iibertragene Hepatitis-Viren und AIDS. Schweiz Zschr GanzheitsMed 996;8(7/8): 325-330.

46 Schreeder MT, Thompson SE, Hadler SC et al. Hepatitis B in homosexual men: Relevance of infection and factors related to transmission. J Infect Dis 1982; 146:7-15.

47 Louria DB, Heusle T, Rose J. The major medical complications of heroin addiction. Ann Int Med 1967;67:1-22.

48 Tabor E. Review of the transmission of hepatitis by clotting factor concentrates. Scand J Haematol 1983;33(Suppl.40):323-328.

49 Aach RD, Lander JJ, Sherman LA et al. Transfusion-transmitted viruses: Interim analysis of hepatitis among transfused and non transfused patients. In: Viral hepatitis. (Eds.: GN Vyas, SN Gohen, R. Schmid) Philadelphia: Franklin, 1978.

50 Fricker HS, Segal S. Narcotic addiction, pregnancy and the newborn. Ann J Dis Child 1978;132:360366.

51 Root-Bemstein RS. Rethinking AIDS. New York: Free press,1993:48.

52 Hdssig A, Kremer H, Liang WX, Stampfli K. Hyperkatabole Krankheiten. Schweiz Zschr GanzheitsMed 1997;9:79-99.

53 Calvano SE Hon-nonal mediation of immune dysfunction following thermal and traumatic injury. Adv Host Defence Mechanism 1986; 6: 111-141.

54 Hdssig A, Kremer H, Lanka St, Liang WX, Stampfli K. AIDS und Auto- Immunitdt. Schweiz Zschr GanzheitsMed 1997;9:219-221.

55 Papadopulos-Eleopulos E, Turner VF, Papadimitriou JM. Is a positive Western Blot proof of HIV infection? BioTechnology 1993; 1 1:696-702.

56 Papadopulos-Eleopulos E, Turner VF, Papadimitriou JM. Has Gallo proved the role of HIV in AIDS? Emergency Med (Australia) 1993;5:71-74.

57 Papadopulos E, Johnson C. Is HIV the cause of AIDS? Interview. Continuum 1997;5:8-19.

58 Lanka S. Fehldiagnose AIDS. Wechselwirkungen 1994; 1 2:48-53.

59 Lanka S. HIV - Realitqt oder Artefakt? Raum und Zeit 1995;77:17-27.

60 Lanka S. HIV - Reality or artefact? Continuum 1995;3/1:4-9.

61 Samgadharan MG, Markham PD. The role of human T-lymphotropic retroviruses in leukemia and AIDS. In: AIDS - acquired immune deficiency syndrome - and other manifestations of HIV infection. (Ed.: GP Worrnser.) Park Ridge NJ: Noyes, 1987:197 198.

62 Westhoff J. Zwischen Hysterie und Abwiegelei. Die ratlose Republik.Bild der Wissenschaft 1985; 1 2:88-90.

63 Weltbevblkerung. Knick in der Kurve. Spiegel 1998; Nr.4: S. 165.

64 WHO. Oral information on behalf of Dr. Brown, WHO deputy chairman ,Global AIDS Program. Geneva, Mdrz 1993.

regimed c/o
Dr. med Heinrich Kremer
Metzendorfer Weg 36
D-21224 Rosengarten (b. Hamburg)



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Red 86 GT


"Fascism should more appropriately be called Corporatism, because it is a merger of State and corporate power."
Benito "Il Duce" Mussolini

Maggiore's controversial book, What if Everything You Thought You Knew About AIDS Was Wrong? questions the most basic medical and scientific findings about the disease.
"The idea that HIV causes AIDS is an idea that has not been proven to be correct or true," she says. "There are many valid, vital reasons to go back and rethink what we've been told."

Activists and many AIDS experts have attacked her for her dissident views, but Maggiore's influence is growing, and her voice has been heard across the country and around the world.

American Foundation for AIDS Research (AmfAR) co-founder Dr. Mathilde Krim fears Maggiore is doing incalculable harm in the fight against AIDS.

"The problem here is that she's spreading the delusion to others without any doubt that she may be wrong," Krim says. "This is terrible. This is what makes me angry."

Path to Dissidence

In 1990, two years after her long-term relationship ended, Maggiore tested positive for HIV in a routine medical exam. She soon learned that her ex-boyfriend had also tested HIV positive. Believing she was terminally ill, she devoted herself to warning others about the dangers of AIDS.

"I encouraged people to take tests. I called them accurate and specific," she says. "I told people that everything added up in the world of AIDS science, and I believed that with my heart."

But a year after she was diagnosed, another HIV test came back indeterminate and a subsequent test was negative.

Frustrated and angry, Maggiore desperately searched for answers. The more she read, the more questions she had. She was shocked to learn that HIV tests measure antibodies, not the virus itself, and that scientists still have questions about the exact process by which HIV causes AIDS.

She discovered the writings of Dr. Peter Duesberg, a controversial virologist at the University of California, Berkeley, who has been saying for years that HIV could not cause AIDS.

"I realized that what I had been taught and what I was teaching other people did not add up," she says. "Many times it was simply wrong."

She became convinced AIDS was not caused by HIV, but by other known immune-suppressing risk factors such as recreational drug use, toxic AIDS treatments, even poverty and malnutrition.

"The diseases we call AIDS can range from chronic yeast infections to certain forms of cancer to certain kinds of pneumonias," she says. "These happen to people who don't test HIV positive."

Mainstream scientists, however, say the evidence is irrefutable. HIV can be found in the blood of almost 100 percent of those diagnosed with epidemic AIDS, and virtually no one without HIV will develop AIDS.

"The evidence that HIV causes AIDS is as good as the evidence that exists that polio is caused by a polio virus and measles by a measles virus," says Krim.

Controversial Decisions

Maggiore knows that without treatment, she has a 95 percent chance of dying from AIDS within the next six years.

But not only has she refused to take anti-HIV drugs she engaged in unprotected sex with her husband, documentary filmmaker Robin Scovill, who knew Maggiore was HIV positive when they became involved.

Shortly after the two became intimate, Maggiore discovered she was pregnant. "First we laughed and then we cried and then we laughed," she remembers. Her son Charlie is now 3 years old, and Maggiore is now pregnant with their second child.

Doctors warn that there is a 25 percent chance her children will also be infected with the virus, because in both pregnancies, she refused to take anti-HIV drugs like AZT.

She argues that the powerful drugs would do more harm than help. "AZT is a drug that disrupts, destroys forming DNA chains in the body, that's the central molecule of life," she says. "I did not want to expose my growing child to toxins during pregnancy."

Like his father, Charlie has never been tested for HIV. Maggiore has chosen not to test her son because she doesn't want to subject him to the stigma of HIV. "I don't need to risk introducing into his life a label that will wrongly describe him as ill when he's not."

She's also made the radical decision to breastfeed Charlie, even though experts say HIV can be transmitted through breast milk.

Private Convictions Gone Public

Her fervent convictions put her in the spotlight.

She met with San Francisco mayor Willie Brown, who was sympathetic to her beliefs. And last summer, Maggiore stepped onto the world stage at the International AIDS Conference in Durban, South Africa. There she met with President Thabo Mbeki, who reportedly became intrigued by the dissidents' views while surfing the Internet.

Protests erupted when Mbeki stunned the world by questioning whether HIV was indeed the cause of the AIDS epidemic devastating his country. The American Foundation for AIDS Research shot back with a full-page ad in The New York Times saying, "HIV is caused by AIDS, to argue otherwise costs lives."

Krim worries that the publicity she has garnered is a step in the wrong direction in the fight against AIDS.

"What she says she has learned," Krim says, " drives people to the conclusion that they can throw away their condoms and stop taking medication."

Convinced She's Beating the Odds

But Maggiore says she's simply trying to encourage people to question conventional ideas about the disease.

"All I'm asking is for people to think about these issues," she says. "I'm providing information that people can use to make informed choices about their life and their health."

Experts say that the incubation period between HIV infection and full blown AIDS is 10 years. Maggiore was infected 11 years ago — and has yet to develop any symptoms of the disease.

Krim says that she is simply a "slow progressor."

But Maggiore remains convinced she's beaten the odds. She is now five months pregnant with her second child.
www.abcnews.com (http://www.abcnews.com)

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Manu Narayan

I saw this freak on 20/20 the other night..

and all i can say is she is dilutional.

She has un-protected sex with her husband, and breast fead her son. And they refuse to get tested...

Now she claims that she shouldn't have to live her life as a dieing person..and i agree...but refusing to take drugs for her HIV is just stupid...and she is converting more people into following her ideals, not taking anything...having un-protected sex....that is just a travesty.

99% of the medical science community will say that HIV is the cause of AIDS.

And people usually don't die of AIDS..they die before they get full blown aids...

I am under the knollege that AIDS is the totall loss of all T-Cells in the body.

HIV slowly dropps your sickness fighting T-cells, till you get something that your body can't fight...like pneumonia, or some other virus.

I red a book called Regarding Nancy...or Go Ask Nancy...i can't remember the first part of the title.

It was the personall journal of a teen with HIV and her life..and death from HIV...she never got full blown AIDS...it was tragic...and YES COSMO...i cried...(and you can bite me if you think i'm gay because i cried over something sad)

I would be wonderful if HIV wasn't the cause of aids...but unfortunately...it is..and we have to deal with reality...not fantasy.

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"The only thing i know is that i don't know"
-Socrates
http://216.105.37.221/images/gif006.gif

I couldn't disagree more, there is a reason Christine is still alive and healthy, it's because she is not taking the toxic aids drugs. It took me years to change my beliefs about aids, and 99% of the medical science community does not agree with the whole aids thing. There are countless doctors and scientists who are coming forward and trying to shed the truth on the issue, it's just that mainstream media will not listen to anybody who does not tout the official line.

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Red 86 GT


"Fascism should more appropriately be called Corporatism, because it is a merger of State and corporate power."
Benito "Il Duce" Mussolini

Originally posted by Scott:
I would be wonderful if HIV wasn't the cause of aids...but unfortunately...it is..and we have to deal with reality...not fantasy.



Yes, thats exactly right. There are downright fabrications in that article.
I believe the word used to describe that article was "crap" from a well respected physician I know.
Its not an attack on you CA, please dont take it that way, it is an attack on the validity and credibility of that article.



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You and I, We are strangers by one chromosome

Hey, I don't care about questioning anything, but I don't consider it crap, some of the most respected doctors and scientists around the world are speaking out against the aids myth. After spending years doing my own research into aids I have come to the same conclusion, HIV is harmless. Immune suppression can be caused by a number of things, unhealthy eating habits, unhealthy lifestyle, environmental pollution, drug use, etc. Even the drug AZT that is given to people diagnosed with HIV with the bullshit antibodies test can kill you. It is one toxic mutha, not something a sickly person needs to be ingesting. Now this is a immensly complicated issue and not one that I can explain on a little message board, there are plenty of ways to find the truth about it, the net is full of info, bookstores carry books about it. It's not very hard to find out about it. Heck, even Magic Johnson has put on weight and lived for a decade after being diagnosed with having antibodies in his blood (which we all do as a immune response). I have heard that he doesn't take the aids drugs.

But when it all comes down to it I don't really care if people think I'm crazy, all I can try to do is help them. I feel liberated not living with the fear of catching aids.


This page has tons of info.. ALOT of info and ALOT of doctors, scientists that agree.. http://www.buildfreedom.com/aidsdir.htm

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Red 86 GT


"Fascism should more appropriately be called Corporatism, because it is a merger of State and corporate power."
Benito "Il Duce" Mussolini

This is an interesting little paragraph from a virologist..

----------------------------

In his own work, German virologist Stefan Lanka has reached the same conclusion: "A virus is an easily definable entity. It’s the very stable product of cells...easy to isolate. To characterize a virus, you have to photograph the isolated particle; then you destroy the virus, characterize the proteins of the virus, and photograph the protein. And you do the same with the genetic material of the virus....This has never ever been done with HIV." 3



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Red 86 GT


"Fascism should more appropriately be called Corporatism, because it is a merger of State and corporate power."
Benito "Il Duce" Mussolini

I am not willing to go 100% that HIV/AIDS are not related and that the drugs are all a sham, but believing it is true simply cause that is what doctors say.

Scott, did you know 99% of everyone believed the world was flat and the sun revolved around us? Did that make them right?

The fact is, there IS a fair amount of evidence to show that our current understanding of the HIV/AIDS relationship is not completely if not false. That should be eough to question it to the point of insisting on further research.

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Manu Narayan

OK I used to be an HIV nurse. (I'm still a nurse but working in another area.)

There are other viruses that are very similar to HIV which cause AIDS. When you look up the definition of AIDS it's very definition is linked with HIV. Therefore, whether someone has AIDS or not comes down to Drs' diagnoses. In some countries, a doctor will say a patient has AIDS after they have been found to be HIV positive and have had one of the associated illnesses. In other countries they have to have had two associated illnesses.

It is true that the ARV's (anti-retro viral) therapies are very toxic. They do have hideous side effects. However, there is no doubt that the death rate has dropped dramatically since the introduction of ARVs. I have nursed people who have been positive for 18 years, and who were at "death's door" prior to being given ARV therapy.

AZT is given to pregnant women who are positive. The results of which are that a huge proportion (nearly 100%) gave birth to negative babies.

In Britain, we only suggest ARV therapy when it is appropriate. You can be HIV positive for years before needing ARV. There are two things we look for, viral load, and CD4 count. If the viral load is high and the CD4 count is low we may suggest ARVs. However, if the viral load is high but the CD4 count is within normal range then we don't suggest ARV.

When ARV's first came out, the American doctors gave them to all HIV positive patients. They have now switched to the British method for several reasons, the main one being the side effects and the impact the drug has on the virus.
Once you start ARV's you are on them for life!!!! And they cost £10,000 per year, which must be about $14,500.

Oh, and a further point to add is this.

I have known people who have died of AIDS who were not gay, were not drug addicts, were not promiscuous. They were unlucky.
One young girl I know who is HIV +, fell for the wrong guy (bisexual drug addict) and paid the price.

There is so much stigma attached to this disease. Why is it important to know how people got it?
People make mistakes in life, do they deserve to pay this price for them?

I hate the whole stigma, and the way people judge HIV positive people. I have nursed many people who have been unable to tell their friends and families about their diagnosis because they know they will be judged. When someone is dying, they shouldn't have to worry about that. One man who died on my ward, died worrying about what was going to be written on his death certificate. That's not right!

Originally posted by Icarus


There is so much stigma attached to this disease. Why is it important to know how people got it?
People make mistakes in life, do they deserve to pay this price for them?



You're kidding, right? I think I know what you're saying, but, honestly, if there's a terrible disease out there, I want to know HOW it's spread. If there's a way to curb its spread, honestly, I don't care who's feelings get hurt, you know?

To anonymously be counselled about how someone became HIV + is one thing. To insist that an individual's private life become public knowledge is another.

Everyone is counselled when they are tested. One of the reasons is to determine how they became positive so that it is possible to contact other people who may be infected by that person. The knowledge learnt from that is how we know the way it's transmitted.

Oh, I don't want it broadcast who's got it and who doesn't. That wasn't what I meant.

I DO want to know HOW the disease is passed on, though.

I was young during the emergance of all of this (I'm 32, now), and I can remember everyone talking about how you get it (and don't get it). Basically, there was going to be the "next wave" or the huge "heterosexual AID's epademic" that was going to kill 40% of the population. It never happened . . .

I don't know exactly WHAT causes it, but, it sounds to me that these people with the HIV virus are in denial. I just don't think that nutrition or somethihg like that causes the condition.

Im surprised nobody else has heard this.

I was in a class with an intelligent girl who did a report on this very thing. A few months ago, doctors announced it, but did not seem to be big news. HIV does not cause AIDs. But there is a reason why many patients have both. Their weakened immun systems allow virus to enter and live within the body. Once HIV has innfected the body, and is not treated, many viruses enter your body. In fact, is is not HIV or AIDs that kills people, it is the other viruses, like the flu, that the body can no longer fight.

Originally posted by D Durden
Oh, I don't want it broadcast who's got it and who doesn't. That wasn't what I meant.

I DO want to know HOW the disease is passed on, though.

I was young during the emergance of all of this (I'm 32, now), and I can remember everyone talking about how you get it (and don't get it). Basically, there was going to be the "next wave" or the huge "heterosexual AID's epademic" that was going to kill 40% of the population. It never happened . . .

I don't know exactly WHAT causes it, but, it sounds to me that these people with the HIV virus are in denial. I just don't think that nutrition or somethihg like that causes the condition.

They are in denial. Poor nutrition does affect the immune system, but does not cause HIV. The virus can only be transmitted person to person. Of that I am 100% sure. I don't know why these people are making claims such as this. It's not going to help future generations at all.

As a further thought about the spread of HIV, the fastest growing population (in Europe and America) of people being diagnosed as HIV + are heterosexual females.
Females can become infected much easier than males due to the biological differences. It's a question of plumbing. ;)

As for killing 40% of the population, well it's not quite done that not in the west anyway. Africa is a whole other ball game. I think when AIDS was first "BIG News" in Europe and America, there was lots of speculation about what would happen in the future. Now things are different. We have lots of information available.

There is a rise in the number of people with TB in England at the moment. TB has been a disease which is associated with HIV since the 80's, so people with TB are advised to be tested. However, it has been found that some people with TB aren't HIV+. This has been put down to two things. An increase in people with compromised immune systems, and the introduction of a sturdier form of TB which has evolved due to the widespread vaccination programs.
It seems to me that the people you are talking about may have picked up on the fact that some of the general population have been found to be more immuno-compromised than before. Diet and lifestyle do affect the immune system. Things like smoking, recreational drugs, alcohol abuse, and poor nutrition will inhibit the immune response to some extent.
However, I must point out that most of the people with TB in England, have been the homeless and some elderly people living in extreme poverty. It seems that those affected have been at the far end of the scale in terms of poor nutrition.
Therefore, I think you are right, these people are trying to put the blame for their infection on other areas. HIV DOES NOT just happen to people. You have to be infected with it.

There are some research studies being carried out, with the purpose of developing a vaccine, on some Thai prostitutes who carry the virus in their vaginas, but are not carrying the virus in their blood. For some reason the virus never crossed the barrier and entered their bloodstream. Which means they will never get sick, but can pass on the virus to others.

The bottom line is, if you practice safe sex, don't share needles, and don't drink other people's blood, you cannot get infected.
Anyone suggesting otherwise is totally deluded. The virus can only be transmitted person to person.

In response to ChaoticThoughts, people don't die of AIDS, they die because of AIDS. So you are right. AIDS affects the immune system so that they cannot fight off disease. It's the disease which is the cause of death. This is why it's a bit dubious when it comes to statistics. The death statistics will be affected by what's put on the death certificate. If the specific illness is put down that's different than recording death "due to AIDS related illness".